Acromioclavicular Joint Injuries



Ovid: Rockwood And Green’s Fractures In Adults

Editors: Bucholz, Robert W.; Heckman, James D.; Court-Brown, Charles M.; Tornetta, Paul
Title: Rockwood And Green’s Fractures In Adults, 7th Edition
> Table of Contents > Section Two – Upper Extremity > 39 – Acromioclavicular Joint Injuries

39
Acromioclavicular Joint Injuries
Leesa M. Galatz
Ronald F. Hollis Jr.
Gerald R. Williams Jr.
INTRODUCTION
The acromioclavicular (AC) joint is commonly involved in
traumatic injuries that affect the shoulder. Treatment of these
injuries has been controversial and continues to evolve to this day.
Most injuries are related to falls onto the shoulder and to repetitive
use of the shoulder, such as heavy labor and athletics. This chapter
focuses primarily on the traumatic aspects of AC disorders and
describes the anatomy, classification, biomechanics, diagnosis, and
treatment of these injuries.
Dislocation of the AC joint, particularly its treatment,
has been a subject of controversy from the earliest medical writings.
Hippocrates1 (460-377 BC) wrote:
Physicians are particularly liable to be deceived in
this accident (for as the separated bone protrudes, the top of the
shoulder appears low and hollow), so that they may prepare as if for
dislocation of the shoulder; for I have known many physicians otherwise
not expert at the art who have done much mischief by attempting to
reduce shoulders, thus supposing it as a case of dislocation.
Galen1 (129-199 AD)
obviously had paid close attention to Hippocrates, because he diagnosed
his own AC dislocation received from wrestling in the palaestra. This
famous physician of the Greco-Roman period treated himself in the
manner of Hippocrates (i.e., tight bandages to hold the projecting
clavicle down while keeping the arm elevated). He abandoned the
treatment after only a few days because it was so uncomfortable. It is
appropriate that one of the earliest reported cases in the literature
was related to sports, because today participation in sports is
certainly one of the most common causes of AC dislocations.

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From the earliest publications through the time of Paul
of Aegina (7th century), dislocations of the AC joint have become
better recognized. Their treatment, however, has remained essentially
unchanged. Hippocrates1 stated that
no impediment, small or great, will result from such an injury. He
further stated that there would be a “tumefaction” or deformity, “for
the bone cannot be properly restored to its natural situation.” This
statement apparently was, has been, and will be received by the
orthopaedic community as a challenge. There is probably not another
joint in the body that has been treated in so many different ways as
the AC joint in attempts to “properly restore” it to “its natural
situation.”
The treatment of AC joint injuries has evolved and
changed as our understanding of the nature of the problem and the
biomechanics of the joint has developed. In 1917, Cadenat18 described the transfer of the coracoacromial ligament, which was later popularized by Weaver and Dunn.152
This remains the most commonly used and successful surgical treatment
we have today for many complete AC dislocations. Surgical treatment was
very common in the 1940s to the 1960s for complete dislocations.144
After further study, the complete dislocations (of the
AC joint) according to older classification systems were broken down
into more detailed groupings depending on the position of the clavicle
and the degree of soft tissue injury.121
Now, treatment addresses the specific pathology involved, and many of
the injuries thought to need treatment in the past are successfully
treated with conservative measures. Treatment remains controversial in
many circumstances, as over the years numerous surgical methods have
been described.
PRINCIPLES OF MANAGEMENT
Mechanism of Injury
Direct force is the most common mechanism of injury and
is produced by the patient falling onto the lateral aspect of the
shoulder with the arm in an adducted position (Fig. 39-1). The force drives the acromion downward and medially. Bearn7
showed that downward displacement of the distal clavicle is primarily
resisted through an interlocking of the sternoclavicular ligaments. If
no fracture occurs, the force first sprains the AC ligaments (a mild
sprain), then tears the AC ligaments (a moderate sprain) and stresses
the coracoclavicular ligament, and finally—if the downward force
continues—tears the deltoid and trapezius muscle attachments from the
clavicle and ruptures the coracoclavicular ligaments (a severe AC
sprain, which completes the dislocation). At this point, the upper
extremity has lost its suspensory support from the clavicle and the
scapula displaces inferiorly.
The mechanism for the inferior dislocation of the
clavicle under the coracoid is thought to be a very severe direct force
onto the superior surface of the distal clavicle, along with abduction
of the arm and retraction of the scapula.97 This type of AC joint dislocation is exceedingly rare.
Classically, the literature indicates that upward
displacement of the clavicle is diagnostic of a complete AC
dislocation. Although there may be a slight upward displacement of the
clavicle by the pull of the trapezius muscle, the characteristic
anatomic feature is actually inferior displacement of the shoulder and
arm. The scapula and attached upper extremity are suspended from the
clavicle primarily by the coracoclavicular ligaments and secondarily
through the AC ligament and the surrounding musculature. Therefore,
when a severe downward force is applied to the point of the shoulder
(assuming the sternoclavicular ligaments do not rupture and the
clavicle does not fracture), the coracoclavicular ligaments rupture.
The suspension system of the scapula and attached upper extremity from
the clavicle is lost. Consequently, the arm displaces inferiorly (Fig. 39-2).
Because the weight of the arm is no longer suspended from the clavicle,
there may be a slight upward pull by the trapezius muscle on the
clavicle. However, the major deformity seen in complete AC dislocation
is a downward displacement of the shoulder.
FIGURE 39-1 The most common mechanism of injury is a direct force that occurs from a fall on the point of the shoulder.
FIGURE 39-2
Anteroposterior radiograph demonstrating a chronic AC joint dislocation
on the left upper extremity. Note that the clavicles are in the same
position bilaterally, and the left scapula is displaced inferiorly.
(From Rockwood CA, Young DC. Disorders of the acromioclavicular joint.
In: Rockwood CA, Matsen F III, eds. The Shoulder. Philadelphia: WB
Saunders, 1990:434.)

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Associated Injuries
Scapulothoracic Dissociation
AC separations may be associated with other injuries
about the shoulder. Scapulothoracic dissociations are characterized by
lateral displacement of the scapula, a severe neurovascular injury, and
an injury to bone (either an AC separation, a displaced clavicle
fracture, or a sternoclavicular disruption). Scapulothoracic
dissociations are usually clinically obvious. Injuries associated with
AC separations, however, may be less obvious. The patient not only will
have pain in the shoulder but also will complain of chest pain and pain
in the periscapular and perithoracic region. Clinical examination
demonstrates the AC deformity as well as marked tenderness in the
periscapular and perithoracic region. An anteroposterior chest
radiograph demonstrates an increased distance between the medial
scapular border and the midline on the affected side compared with the
unaffected side, as well as perhaps a pleural effusion. Magnetic
resonance imaging of the thorax demonstrates increased signal in the
periscapular and perithoracic muscles in addition to a pleural effusion.
Fractures
Fractures associated with AC separations may include
fractures of the clavicle, the acromion process, the coracoid process,
and the sternoclavicular joint. Wurtz and colleagues157
reported 4 patients with a fracture of the middle third of the clavicle
and dislocation of the AC joint. Various treatment methods were used
and achieved good results in all four patients with 1- to 3-year
follow-up. Barber6 reported a
patient with a type IV AC joint injury associated with a contralateral
pneumothorax and an ipsilateral pulmonary contusion.
Brachial Plexus Abnormalities
Meislin and colleagues98
have reported a patient who developed a brachial plexus neuropraxia 8
years after sustaining a type III AC separation. The patient responded
well to coracoclavicular stabilization. Brachial plexus injuries
associated with AC separations are not common. Sturm and Perry,137 in a review of 59 patients with brachial plexus injuries, identified two patients with AC separations.
Coracoclavicular Ossification
Coracoclavicular ossification has been referred to as both ossification and calcification. Urist146
demonstrated that bone does indeed form in the coracoclavicular
interval. The calcification can be formed heterotopically around the
area of injury, or it can form a bridge between the coracoid and the
clavicle. Usually, it has no effect on the functional outcome.
Osteolysis of the Distal Clavicle
Osteolysis of the distal clavicle may follow an acute
injury or may occur in persons who have repeated stress on the
shoulder. Madsen88 reported seven
patients with the rare complication known as posttraumatic osteolysis
of the distal clavicle. He identified eight cases in the literature at
that time (1963), the first of which was reported by Werder in 1950.
Cahill19 reported 46 patients who
were athletes, none of whom had an acute injury, but 45 of whom lifted
weights as part of their training. He used technetium bone scans and a
35-degree cephalic tilt radiographic view to help make the diagnosis.
Several authors have reported this condition in women.92,99,112
The radiographic findings are osteoporosis, osteolysis,
and tapering of the distal clavicle. Usually, bony changes do not occur
in the acromion. Changes usually occur only in the injured shoulder. If
changes are noted in both shoulders, then other conditions should be
considered, such as rheumatoid arthritis, hyperparathyroidism, and
scleroderma. The differential diagnosis of a lesion in one shoulder
should include Gorham’s massive osteolysis, gout, and a neoplasm such
as multiple myeloma. Microscopic studies of the distal clavicle have
been reported by Murphy and coworkers104 and Madsen.88 They described demineralization, subchondral cysts, and erosion of the distal clavicle. Griffiths and Glucksman56 performed a biopsy 8 months after injury that showed patches of necrotic and reactive woven bone.
History and Physical Examination
As with any orthopaedic evaluation, a thorough history
should be obtained detailing the mechanism of injury as well as a
complete discussion of patient symptoms. A complete evaluation of all
injured extremities should be documented including a neurovascular
examination. When AC joint injury is suspected, the patient should be
examined, whenever possible, in the standing or sitting position. The
weight of the arm stresses the AC joint and makes a deformity more
apparent.
Type I Injury
In a type I injury, there is minimal to moderate
tenderness and swelling over the AC joint without palpable displacement
of the joint. Usually there is only minimal pain with arm movements.
Tenderness is not present in the coracoclavicular interspace.
Type II Injury
With subluxation of the AC joint, moderate to severe
pain is noted at the joint. If the patient is examined shortly after
injury, the outer end of the clavicle may be noted to be slightly
superior to the acromion. Motion of the shoulder produces pain in the
AC joint. With gentle palpation, the lateral end of the clavicle is
unstable in the anterior-posterior direction. If the midclavicle is
grasped and the acromion stabilized, posterior-anterior motion of the
clavicle in the horizontal plane can be detected. There should be
little, if any, instability in the vertical plane. Tenderness is also
noted when the physician palpates anteriorly in the coracoclavicular
interspace.
Type III Injury
The patient with type III injury, complete dislocation
of the AC joint, characteristically presents with the upper extremity
held adducted close to the body and supported in an elevated position
to relieve the pain in the AC joint. The shoulder complex is depressed
when compared with the normal shoulder. The clavicle may be prominent
enough to tent the skin (Fig. 39-3). Moderate pain is the rule, and any motion of the arm, particularly abduction, increases the pain.
Tenderness is noted at the AC joint, the
coracoclavicular interspace, and along the superior aspect of the
lateral fourth of the clavicle. The entire length of the clavicular
shaft should be palpated to detect an associated clavicle shaft
fracture. The lateral clavicle is unstable in both the horizontal and
vertical

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planes.
The key to the diagnosis of a type III injury is that the defect can be
reduced with upward pressure under the elbow. A reducible injury is
differentiated from a type IV or V injury, which cannot be reduced (see
below).

FIGURE 39-3
This patient has a complete type III dislocation of the left AC joint.
The left shoulder is drooping, and there is prominence of the left
distal clavicle. (From Rockwood CA, Young DC. Disorders of the AC
joint. In: Rockwood CA, Matsen F III, eds. The Shoulder. Philadelphia:
WB Saunders, 1990:425.)
Type IV Injury
The patient with a type IV injury has essentially all
the clinical findings of a type III injury. In addition, examination of
the seated patient from above reveals that the outline of the displaced
clavicle is inclined posteriorly compared with the uninjured shoulder.
The clavicle usually is displaced so severely posteriorly that it
becomes “buttonholed” through the trapezius muscle and tents the
posterior skin (Fig. 39-4). Consequently,
motion of the shoulder is more painful than in a type III injury. The
AC joint cannot be reduced manually in this situation. The
sternoclavicular joint should always be examined for an associated
anterior dislocation.126
FIGURE 39-4
Patient with type IV AC joint injury. Note that the distal end of the
clavicle is displaced posteriorly back into and through the trapezius
muscle. (From Rockwood CA, Young DC. Disorders of the acromioclavicular
joint. In: Rockwood CA, Matsen F III, eds. The Shoulder. Philadelphia:
WB Saunders, 1990:446.)
FIGURE 39-5 A photograph of a type V AC joint dislocation demonstrating the superior displacement of the clavicle relative to the shoulder.
Type V Injury
The type V injury is an exaggeration of the type III
injury in which the distal end of the clavicle appears to be grossly
superiorly displaced and tenting the skin (Fig. 39-5).
This apparent upward displacement is the result of downward
displacement of the upper extremity. The patient has more pain than
with a type III injury, particularly over the distal half of the
clavicle. This is secondary to the extensive muscle and soft tissue
disruption from the clavicle that occurs with this injury. The distal
clavicle is subcutaneous and cannot be manually reduced. Occasionally,
there is so much inferior displacement of the upper extremity that the
patient will develop symptoms of traction on the brachial plexus.
Type VI Injury
Type VI injuries are very rare. The superior aspect of
the shoulder has a flat appearance, as opposed to the rounded contour
of the normal shoulder. With palpation, the acromion is prominent, and
there is a definite inferior stepdown to the superior surface of the
coracoid process. Because of the amount of trauma required to produce a
subcoracoid dislocation of the clavicle, there may be associated
fractures of the clavicle and upper ribs or injury to the upper roots
of the brachial plexus. These associated injuries may produce so much
swelling of the shoulder that the disruption of the AC joint may not
initially be recognized. Vascular injuries secondary to the dislocation
were not present in the patients presented by McPhee,97 Schwarz and Kuderna,130 and Gerber and Rockwood.54 However, all the adult cases reported by McPhee102 and Gerber and Rockwood54 had transient paresthesias before reduction of the dislocation. After reduction, the neurologic deficits resolved.
Imaging and Other Diagnostic Studies
Good-quality radiographs of the AC joint require one third to one half the x-ray penetration required to image the glenohumeral

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joint. Radiographs of the AC joint taken using routine shoulder
technique will be overpenetrated (i.e., dark), and small fractures may
be overlooked. Therefore, the x-ray technician must be specifically
requested to take radiographs of the “AC joint” rather than the
“shoulder.”

Anteroposterior Views
Routine anteroposterior views should be obtained with
the patient standing or sitting, with the back against the x-ray
cassette and the arms hanging unsupported at the side. Because of
significant individual variation in AC joint anatomy and because the
coracoclavicular interspace will vary with the angle of the x-ray beam
and with the distance between the beam and the patient, both AC joints
should be imaged simultaneously on one large (14- × 17-inch) cassette.
Large patients with shoulders too broad to be visualized on a single
cassette should have radiographs made with two smaller (10- × 12-inch)
cassettes using identical technique.
The difficulty in evaluating AC joint injuries lies in
the fact that with this projection, the distal clavicle and acromion
are superimposed on the spine of the scapula. Subtle fractures of the
distal clavicle are easily missed. Zanca159
noted this during a review of 1000 radiographs of patients with
shoulder pain. Therefore, he recommended a 10 to 15 degree cephalic
tilt view to project an unobscured image of the joint (Fig. 39-6).
This view is now routinely used in the evaluation of AC joint injuries
and is particularly useful when there is suspicion of a small fracture
or loose body on routine views (Fig. 39-7).
FIGURE 39-6
Position of the patient for the Zanca view—a 10- to 15-degree cephalic
tilt of the standard view for the AC joint. (From Rockwood CA, Young
DC. Disorders of the acromioclavicular joint. In: Rockwood CA, Matsen F
III, eds. The Shoulder. Philadelphia: WB Saunders, 1990:428.)
Lateral Views
As with any musculoskeletal injury, a radiograph in one
plane is not sufficient to classify an AC joint injury. An axillary
lateral view should be taken of the injured shoulder when an AC
dislocation is suspected. The cassette should be placed on the superior
aspect of the shoulder and medial enough to expose as much of the
lateral third of the clavicle as possible. This will reveal any
posterior displacement of the clavicle as well as any small fractures
that may have been missed on the anteroposterior view.
Stryker Notch View
A variant of an AC joint injury involves a fracture of
the coracoid process. This injury should be suspected when there is an
AC joint dislocation on the anteroposterior projection, but the
coracoclavicular distance is normal, or equal to that on the opposite,
uninvolved side. A Stryker notch view taken appropriately puts the
coracoid in profile and is the best view for evaluating this injury.
This is performed with the patient supine and the arm elevated over the
head with the palm behind the head. The humerus must be parallel to the
longitudinal axis of the body, with the elbow pointed straight toward
the ceiling (Fig. 39-8).
Other Modalities
Schmid and Schmid129
reported the use of ultrasonography in the diagnosis of 22 cases of
type III AC dislocation. Ultrasound examination demonstrated visible
instability of the distal clavicle, incongruity of the joint, hematoma
formation, or visible ligament remnants in all cases. However, in spite
of the advent of such sophisticated imaging modalities as
ultrasonography, computed tomography (CT), and magnetic resonance
imaging, plain radiography continues to be the most readily available,
cost-effective method for routine investigation of injuries to the AC
joint.
Diagnosis and Classification
Diagnosis
Normal Joints. The width and configuration of the AC
joint in the coronal plane may vary significantly from individual to
individual. This should be remembered so that a normal variant is not
mistaken as an injury. In a study of 100 radiographs of normal
shoulders, Urist146 found that
nearly half (49%) of the AC joints were inclined superolateral to
inferomedial, with the articular surface of the clavicle overriding the
acromion; 27% were vertical, and 3% were inclined superomedial to
inferolateral, with the articular surface of the clavicle underriding
the acromion. Another 21% of the joints were incongruent, with the
clavicle lying either superior or inferior to the acromial articular
surface.
The normal width of the AC joint in the coronal plane is 1 to 3 mm.159 Petersson and Redlund-Johnell115
measured AC joint width radiographically in 151 normal individuals and
drew several conclusions: the AC joint space normally diminishes with
increasing age, a joint space of 0.5 mm in a patient older than 60
years is conceivably normal, and a joint space of greater than 7 mm in
men and 6 mm in women is pathologic.
The coracoclavicular interspace also exhibits
significant individual variation. The average distance between the
clavicle and the coracoid process ranges from 1.1 to 1.3 cm.6 An increase in the coracoclavicular distance of 50% over the normal side

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signifies a complete AC dislocation.6 Complete dislocation has been seen with as little as a 25% increase in the coracoclavicular distance.

FIGURE 39-7 An explanation of why the AC joint is poorly visualized on routine shoulder x-rays. A.
This routine anteroposterior view of the shoulder shows the
glenohumeral joint well. However, the AC joint is too dark to
interpret, because that area of the anatomy has been overpenetrated by
the x-ray technique. B. When the exposure
usually used to take the shoulder radiographs is decreased by two
thirds, the AC joint is well visualized. However, the inferior corner
of the AC joint is superimposed on the acromion process. C. Tilting the tube 15 degrees upward provides a clear view of the AC joint.
FIGURE 39-8
Technique for taking the Stryker notch view to demonstrate fractures of
the base of the coracoid. The patient is supine with a cassette placed
posterior to the shoulder. The humerus is flexed approximately 120
degrees so the patient’s hand can be placed on top of the head. The
x-ray beam is directed 10 degrees superior. (From Rockwood CA, Young
DC. Disorders of the acromioclavicular joint. In: Rockwood CA, Matsen F
III, eds. The Shoulder. Philadelphia: WB Saunders, 1990:433.)
Type I Injury. In a type I injury, the radiographs of
the AC joint are normal, except for mild soft tissue swelling, as
compared with the uninjured shoulder. There is no widening, no
separation, and no deformity.
Type II Injury. In a type II injury, the lateral end of
the clavicle may be slightly elevated. The AC joint, when compared with
the normal side, may appear to be widened. The widening probably is the
result of a slight medial rotation of the scapula and slight posterior
displacement of the clavicle by the pull of the trapezius muscle. The
coracoclavicular space of the injured shoulder is the same as that of
the normal shoulder.
Type III Injury. In obvious cases of complete AC
dislocations, the joint is totally displaced. The lateral end of the
clavicle is displaced completely above the superior border of the
acromion and the coracoclavicular interspace is significantly (25% to
100%) greater than in the normal shoulder (Fig. 39-9). Fractures may be noted involving the distal clavicle or the acromion process.
Rarely, complete AC dislocation will be accompanied by a
fracture of the coracoid process rather than by disruption of the
coracoclavicular ligaments. Although the fracture of the coracoid

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process
is difficult to visualize on routine radiographs, its presence should
be suspected because of the presence of a complete AC separation and a
normal coracoclavicular distance, as compared with the uninjured
shoulder. The best special view for visualizing the coracoid fracture
is the Stryker notch view (Fig. 39-10).
The technique for obtaining this view is described above. A few unusual
injury patterns uncommonly occur and are variations of type III
injuries. Most often, complete separation of the articular surfaces of
the distal clavicle and acromion is accompanied by complete disruption
of the AC and coracoclavicular ligaments. Children and adolescents
usually sustain a variant of complete AC dislocation. Radiographs
reveal displacement of the distal clavicular metaphysis superiorly with
a large increase in the coracoclavicular interspace. These injuries are
most often Salter-Harris type I or II injuries in which the epiphysis
and the intact AC joint remain in their anatomic locations while the
distal clavicular metaphysis is displaced superiorly through a dorsal
rent in the periosteal sleeve (Fig. 39-11).11,30,40,43,66
The lateral epiphysis of the clavicle is barely visible because it is
thin and appears and fuses over a short time period at approximately 19
years of age.

FIGURE 39-9
X-ray appearance of a grade III injury. Not only is the right AC joint
displaced compared with the left, but, more significantly, notice the
great increase in the coracoclavicular interspace on the injured right
shoulder compared with the normal left shoulder.
FIGURE 39-10
Radiographs of a patient with a type III variant injury involving the
AC joint and a fracture of both the base and the tip of the coracoid. A. An anteroposterior radiograph of the injured right side. The coracoid injury is not visualized. B.
A radiograph of the uninjured left side demonstrating that the
coracoclavicular distance is equal on the injured and unaffected sides.
C. An axillary view shows the tip fracture, but the fracture at the base is not easily detected. D. The West Point view clearly shows the fracture at the tip of the coracoid process. (continues)

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FIGURE 39-10 (continued) E. The Stryker notch view clearly shows the fracture at the base of the coracoid. F. Nonoperative treatment in this case led to an excellent result as evidenced by full overhead elevation. G. The patient regained near-normal internal rotation.
Eidman and coworkers40
reported on 25 AC injuries in children treated surgically. In all
patients younger than 13 years of age, there was a lateral
Salter-Harris clavicular fracture rather than a true AC dislocation.
The importance of recognizing this injury is that the intact
coracoclavicular ligaments remain attached to the periosteal sleeve.
Nonoperative management most often results in healing of the clavicular
fracture and thus reestablishment of the integrity of the
coracoclavicular ligaments. Those authors who recommend surgical repair
in selected instances emphasize the importance of repairing the dorsal
rent in the periosteal sleeve.40,43
A second variation of the type III injury involves
complete separation of the AC articular surfaces combined with a
fracture of the coracoid process.20,76,91
This is an extremely uncommon injury. In most cases the
coracoclavicular ligaments have remained intact and attached to the
displaced coracoid process fracture, which most often occurs through
the base. There are only two reported cases of complete AC
separation—coracoclavicular ligament disruption and coracoid process
fracture.150,154 According to the authors, the mechanism of injury for this “triple lesion” is a simultaneous blow to the acromion and forcible

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elbow flexion against resistance. In both of these reported cases, the patients underwent operative repair.

FIGURE 39-11 A.
In children and adolescents, the distal clavicular physis lies medial
to the AC capsular reflection. Injuries in this age group are often
type II Salter-Harris fractures involving the physis rather than AC
dislocations. B. The coracoclavicular
ligaments remain attached to the intact periosteal sleeve while the
medial clavicular fragment displaces through a dorsal periosteal rent.
Both operative and nonoperative methods of treatment
have been described for combined AC dislocation and coracoid process
fracture with intact coracoclavicular ligaments. Results seem to be
similar in both groups. Therefore, most authors recommend nonoperative
treatment. Most often, the coracoid process fracture is
extra-articular. However, we have encountered instances in which the
coracoid fragment contains a significant portion of the glenoid fossa.
The conjoined tendon rotates the coracoid process and glenoid
inferolaterally and can result in substantial articular displacement.
In this situation, open reduction and internal fixation may be
necessary and is predicated on the amount of displacement of the
articular fragment (Fig. 39-12).
FIGURE 39-12 Coracoid fracture with intra-articular extension. A. Anteroposterior radiograph showing the fracture through the coracoid. B. A CT scan showing the glenoid displacement necessitating open reduction and internal fixation of the glenoid.
Type IV Injury. Although the radiographic findings
associated with a type IV injury include a relative upward displacement
of the clavicle from the acromion and an increase in the
coracoclavicular interspace, the most striking feature is the posterior
displacement of the distal clavicle, as seen on the axillary lateral
radiograph (see Fig. 39-13). In patients with
heavy, thick shoulders or in patients with multiple injuries in whom an
axillary lateral view of the shoulder or a scapular lateral
radiographic view cannot be taken, a CT scan may be of great value in
helping to confirm clinical suspicions of a posteriorly dislocated AC
joint.
Type V Injury. The characteristic radiographic feature
of type V injuries is a marked increase (100% to 300%) in the
coracoclavicular interspace. The clavicle appears to be grossly
displaced superiorly away from the acromion (Fig. 39-14).
However, radiographs reveal that the clavicle on the injured side is
actually at approximately the same level as the clavicle on the normal
side, and the scapula is displaced inferiorly.
Type VI Injury. There are two types of inferior AC
dislocation: subacromial and subcoracoid. In the subacromial type,
radiographs reveal a decreased coracoclavicular distance (i.e., less
than the normal side), and the distal clavicle is in a subacromial
location. The subcoracoid dislocation is characterized by a reversed
coracoclavicular distance, with the clavicle displaced inferior to the
coracoid process (Fig. 39-15). Because this injury

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usually is the result of severe trauma, it often is accompanied by multiple other fractures of the clavicle and ribs.

FIGURE 39-13 Type IV posterior dislocation of the AC joint. A. Axillary lateral radiograph of the right shoulder. B. Axillary view with the distal clavicle and acromion outlined.
Classification
Acromioclavicular joint injuries are best classified
according to the extent of damage inflicted by a given force. However,
unlike other joints, the differential diagnosis of sprains of the AC
joint is based on the severity of injury sustained by the capsular
ligaments (AC ligaments) and extracapsular ligaments (coracoclavicular
ligaments), as well as the supporting musculature (deltoid and
trapezius muscles). Therefore, injuries to the AC joint are graded
according to the amount of injury to the AC and coracoclavicular
ligaments. Injuries in this anatomic area have always been referred to
as “AC joint injuries,” although the injuries have varying degrees of
disruption between the scapula and the clavicle, not limited to the one
particular joint.
FIGURE 39-14
An anteroposterior radiograph of a type V dislocation shows the marked
increase in the coracoclavicular interspace. The clavicle appears to be
grossly displaced away from the acromion.
The strength of any classification system depends on its
ability to predict prognosis or the need for surgical intervention.
Rockwood’s group154 developed the most widely accepted classification system, based on the original work of Tossy et al.144
in 1963. It is an expanded, accurate classification system based on the
anatomic severity of the injury. The modified classification is
described below and is summarized in Table 39-1 and illustrated in Figure 39-16.
FIGURE 39-15
Type VI AC dislocation. The distal end of the left clavicle is in the
subcoracoid position. The high-energy trauma causing this injury is
evidenced by the bilateral chest tubes in this patient. (From Rockwood
CA, Young DC. Disorders of the acromioclavicular joint. In: Rockwood
CA, Matsen F III, eds. The Shoulder. Philadelphia: WB Saunders,
1990:447. Courtesy of R.C. Erickson and D. Massillion.)

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TABLE 39-1 Modified Acromioclavicular Joint Injuries Classification

Type

AC Joint

AC Ligament

CC Ligament

Deltoid and Trapezius Muscles

I

Intact

Sprain

Intact

Intact

II

Displaced

Torn

Sprain/Intact

Intact

III

Disrupted

Torn

Torn

Usually intact

IV

Disrupted

Torn

Torn

Detached

V

Disrupted

Torn

Torn

Detached

IV

Inferior displacement of clavicle (extremely rare)

Type I. A mild force to the point of the shoulder
produces a minor strain to the fibers of the AC ligaments. The
ligaments remain intact, and the AC joint remains stable.
Type II. A moderate force to the point of the shoulder
is severe enough to rupture the ligaments of the AC joint. The distal
end of the clavicle is unstable in the horizontal plane (i.e.,
anteroposterior), but vertical (i.e., superoinferior) stability is
preserved by virtue of the intact coracoclavicular ligament. The
scapula may rotate medially, producing a widening of the AC joint.
There may be a slight, relative upward displacement of the distal end
of the clavicle secondary to stretching of the coracoclavicular
ligaments.
Type III. A severe force is applied to the point of the
shoulder which tears the AC and coracoclavicular ligaments resulting in
a complete AC dislocation. The distal clavicle appears to be displaced
superiorly as the scapula and shoulder complex droop inferomedially.
Radiographic findings include a 25% to 100% increase in the
coracoclavicular space in comparison to the normal shoulder.121
Type IV. Posterior dislocation of the distal end of the
clavicle, or a type IV AC dislocation, is relatively rare. The clavicle
is posteriorly displaced into or through the trapezius muscle as the
force applied to the acromion drives the scapula anteriorly and
inferiorly. Posterior clavicular displacement may be so severe that the
skin on the posterior aspect of the shoulder becomes tented. The
literature concerning posterior AC dislocations consists mostly of
small series and case reports.5,64,89,107,135 Some5,89,135 refer to this injury as a “posterior dislocation of the clavicle,” and others64,107 prefer the term “anterior dislocation of the AC joint.”
Type V. Type V AC dislocation is a markedly more severe
version of the type III injury. The distal clavicle has been stripped
of all its soft tissue attachments (i.e., AC ligaments,
coracoclavicular ligament, and the deltotrapezius muscle attachments)
and lies subcutaneously at the displaced AC joint. When combined with
superior displacement of the clavicle owing to unopposed pull of the
sternocleidomastoid muscle, the severe downward droop of the extremity
produces a marked disfiguration of the shoulder. Radiographically, the
coracoclavicular space is increased greater than 100% in comparison to
the opposite, normal shoulder.121
Type VI. Inferior dislocation of the distal clavicle, or type VI AC dislocation, is an exceedingly rare injury.54,97,124 Gerber and Rockwood’s54
series of three patients is the largest one reported in the literature.
The injury is often the result of severe trauma and is frequently
accompanied by multiple injuries. The mechanism of dislocation is
thought to be severe hyperabduction and external rotation of the arm,
combined with retraction of the scapula. The distal clavicle occupies
either a subacromial or a subcoracoid location.
In all reported cases of subcoracoid dislocation, the
clavicle has become lodged behind an intact conjoined tendon. The AC
ligaments are disrupted in either a subacromial or subcoracoid
dislocation. The coracoclavicular ligament, however, is intact in a
subacromial dislocation and completely disrupted in a subcoracoid
dislocation. Likewise, the integrity of the deltoid and trapezius
muscle attachments depends on the degree of clavicular displacement.
Incidence
Rowe and Marble123
retrospectively reviewed the medical records of the Massachusetts
General Hospital and found 52 AC joint injuries among 1603 shoulder
girdle injuries. Most occurred in the second decade of life. Thorndike
and Quigley141 reported AC joint
involvement in 223 of 578 athletes with shoulder injuries. AC injuries
are among the most common injuries affecting hockey and rugby players.31,35 AC dislocation is more common in males (5:1 to 10:1) and is more often incomplete than complete (approximately 2:1).
SURGICAL AND APPLIED ANATOMY AND COMMON SURGICAL APPROACHES
Surgical and Applied Anatomy
The AC joint is a diarthrodial joint located between the
lateral end of the clavicle and the medial margin of the acromion
process of the scapula. The articular surfaces initially are hyaline
cartilage. A fibrocartilaginous disk of varying size and shape exists
inside the joint. Viewed from the anterior-posterior direction, the
inclination of the joint may be almost vertical, or it may be inclined
from downward medially, with the clavicle overriding the acromion by an
angle as large as 50 degrees (Fig. 39-17).
There may be an underriding type of inclination, with the clavicle
facet under the acromion process. The articular surface of the clavicle
overrides the articular surface of the acromion approximately 50% of
the time, and the articular surfaces are incongruent.
There are two types of fibrocartilaginous
intra-articular disks—complete and partial (meniscoid). The disk varies
greatly in size and shape.33 With age, the meniscus undergoes degeneration until it is essentially no longer functional beyond the fourth decade.33,114,125 The nerve supply to the AC joint is from branches of the axillary, suprascapular, and lateral pectoral nerves.
Acromioclavicular Ligaments
The AC joint is surrounded by a thin capsule reinforced
above, below, anteriorly, and posteriorly by the superior, inferior,
anterior,

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and posterior AC ligaments (Fig. 39-18).
The fibers of the superior AC ligament, which is the strongest of the
capsular ligaments, blend with the fibers of the deltoid and trapezius
muscles, which are attached to the superior aspect of the clavicle and
the acromion process. These muscle attachments are important in that
they strengthen the weak and thin ligaments, thereby adding stability
to the AC joint. The AC ligaments stabilize the joint in an
anteroposterior direction (the horizontal plane).32,125,146
Recent studies have shown the distance from the lateral clavicle to the
insertion of the superior AC ligament and capsule to range from 5.2 to
7 mm in women and approximately 8 mm in men, much less than previously
thought.13,130 An AC resection that extends medial to the capsular insertion leads to instability in the horizontal plane.12

FIGURE 39-16 Schematic drawings of the classification of ligamentous injuries to the AC joint. A.
In the type I injury, a mild force applied to the point of the shoulder
does not disrupt either the AC or the coracoclavicular ligaments. B.
A moderate to heavy force applied to the point of the shoulder will
disrupt the AC ligaments, but the coracoclavicular ligaments remain
intact (type II). C. When a severe force
is applied to the point of the shoulder both the AC and the
coracoclavicular ligaments are disrupted (type III). D.
In a type IV injury, not only are the ligaments disrupted, but the
distal end of the clavicle is also displaced posteriorly into or
through the trapezius muscle. E. A violent
force applied to the point of the shoulder not only ruptures the AC and
coracoclavicular ligaments but also disrupts the muscle attachments and
creates a major separation between the clavicle and the acromion (type
V). F. This is an inferior dislocation of
the distal clavicle in which the clavicle is inferior to the coracoid
process and posterior to the biceps and coracobrachialis tendons. The
AC and coracoclavicular ligaments are also disrupted (type VI).

P.1222
FIGURE 39-17
Variations of the inclination of the AC and the sternoclavicular
joints. (Redrawn from DePalma AF. Surgery of the Shoulder.
Philadelphia: JB Lippincott, 1973.)
FIGURE 39-18 Normal anatomy of the AC joint.
Coracoclavicular Ligament
The coracoclavicular ligament is a very strong, heavy
ligament whose fibers run from the outer, inferior surface of the
clavicle to the base of the coracoid process of the scapula. The
coracoclavicular ligament has two components: the conoid and the
trapezoid ligaments (see Fig. 39-18). A bursa
may separate these two portions of the ligament. The trapezoid ligament
measures from 0.8 to 2.5 cm in length and from 0.8 to 2.5 cm in width.
The conoid ligament varies from 0.7 to 2.5 cm in length and from 0.4 to
0.95 cm in width.125 The distance from the lateral clavicle to the lateral most fibers of the trapezoid ligament measures as little as 10 mm.13,62,63,120
The conoid ligament, the more medial of the two, is cone
shaped, with the apex of the cone attaching on the posteromedial side
of the base of the coracoid process. The base of the cone attaches onto
the conoid tubercle on the posterior undersurface of the clavicle. The
conoid tubercle is located at the apex of the posterior clavicular
curve, which is at the junction of the lateral third of the flattened
clavicle with the medial two thirds of the triangular shaft.
The trapezoid ligament arises from the coracoid process,
anterior and lateral to the attachment of the conoid ligament. This is
just posterior to the attachment of the pectoralis minor tendon. The
trapezoid ligament extends superiorly to a rough line on the
undersurface of the clavicle. This line extends anteriorly and
laterally from the conoid tubercle.
Biomechanics
The biomechanics of the AC joint involve static
(ligamentous) stability, dynamic stability, and AC joint motion. This
has been a topic of research for many years, and recently more
sophisticated techniques in biomechanic research have elucidated the
role of the various structures about the joint.
The only connection between the upper extremity and the
axial skeleton is through the clavicular articulations at the AC and
sternoclavicular joints. Bearn7
stressed the importance of the sternoclavicular ligaments in supporting
the distal end of the clavicle. Through anatomic dissections and
selective divisions of the sternoclavicular ligaments, he demonstrated
how these ligaments prevent downward displacement of the distal end of
the clavicle. Hence, in the erect position, the strong sternoclavicular
ligaments support the clavicles out, away from the body, like the wings
off the body of an airplane. Furthermore, just as the jet engines are
suspended from the underside of the wings, the upper extremities are
suspended from the distal clavicles through the coracoclavicular
ligament. Thus, the coracoclavicular ligament is the prime suspensory
ligament of the upper extremity.
AC joint stability is maintained predominantly by the
surrounding ligamentous structures, specifically the coracoclavicular
ligaments (conoid and trapezoid) and the AC capsule and ligaments.
Urist146 demonstrated
that the distal clavicle could be completely dislocated anteriorly and
posteriorly away from the acromion process following excision of the AC
joint capsule. Only after the coracoclavicular ligaments were
transected did vertical displacement of the clavicle relative to the
acromion occur (Fig. 39-19). Fukuda and colleagues48
performed load-displacement tests with a fixed displacement after
sequential ligament sectioning in order to determine individual
contributions of the various

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ligaments
to AC stability. The contribution of the AC, trapezoid, and conoid
ligaments was determined at small and large displacements. At small
displacements, the AC ligaments were the primary restraint to both
posterior (89%) and superior (68%) translation of the clavicle—the most
common failure patterns seen clinically. At large displacements, the
conoid ligament provided the primary restraint (62%) to superior
translation, while the AC ligaments remained the primary restraint
(90%) to posterior translation. The trapezoid ligament served as the
primary restraint to AC joint compression at both large and small
displacements.

FIGURE 39-19 The importance of the AC and coracoclavicular ligaments for stability of the AC joint, demonstrated in a fresh cadaver. A.
With the muscles and AC capsule and ligaments resected and with the
coracoclavicular ligaments intact, the clavicle can be displaced
anteriorly, as shown, or posteriorly from the articular surface of the
acromion. B. However, because the coracoclavicular ligaments are intact, the clavicle cannot be displaced significantly upward. C.
Following the transection of the coracoclavicular ligaments, the
clavicle can be displaced completely above the acromion process. This
suggests that the horizontal stability of the AC joint is accomplished
by the AC ligaments, and vertical stability is obtained through the
coracoclavicular ligaments.
Studies have demonstrated that the trapezoid ligament
has a greater role in resistance to posterior displacement of the
clavicle and the conoid has a greater role in anterior displacement.82,133 The role of the AC joint capsule and ligaments has been studied extensively with respect to distal clavicle resection.15,32,45,46
Posterior abutment of the clavicle against the acromion is avoided with
only 5 mm of bone removal. This preserves the capsule and ligaments,
maintaining anteroposterior stability of the AC joint. Larger
resections have been shown to result in excessive posterior translation.12,75 Together, these experiments have led to the following conclusions:
  • The horizontal stability is controlled by the AC ligament.
  • The vertical stability is controlled by the coracoclavicular ligaments.
These statements are obviously oversimplifications. The
motion and translations that occur at the AC joint are likely to be
much more complicated than biplanar. Further work is clearly needed in
this area.
The coracoclavicular ligament helps to couple
glenohumeral abduction and flexion to scapular rotation on the thorax.
Full overhead elevation cannot be accomplished without combined and
synchronous glenohumeral and scapulothoracic motion.22,70,73
As the clavicle rotates upward, it dictates scapulothoracic rotation by
virtue of its attachment to the scapula—the conoid and trapezoid
ligaments.
Motion of the Acromioclavicular Joint
Motion of the AC joint has been a subject of debate. The
clavicle rotates upward 40 to 50 degrees with elevation of the shoulder.
Rockwood et al.122
showed that there was only 5 to 8 degrees of rotation of the clavicle
relative to the acromion. Although the clavicle rotates 40 to 50
degrees during full overhead elevation, this rotation is combined with
simultaneous scapular rotation rather than with pure AC joint motion.
This “synchronous scapuloclavicular” motion was originally described by
Codman22 and more recently by Flatow.45
Common Surgical Approaches
The patient is positioned in a beach chair configuration with the back of the table 15 to 20 degrees short of the vertical

P.1224

position. It is important to make sure that all potential points of
pressure are well padded. A small roll or a 1 liter intravenous fluid
bag is placed under the ipsilateral scapula to bring the shoulder
forward. This allows good access to the superior aspect of the
shoulder. The skin incision is approximately 6 cm in length and is made
in Langer skin lines 2 to 3 cm medial to the AC joint. Full-thickness
subcutaneous flaps are developed medially and laterally to expose the
deltoid and trapezius aponeuroses the AC joint and the lateral 2 to 3
cm of the distal clavicle.

Electrocautery is used to incise the periosteum and
deltotrapezius fascia parallel to the longitudinal axis of the clavicle
in a medial-lateral orientation at the junction between the anterior
one third and the posterior two thirds of the lateral clavicle (see Fig. 39-24).
The incision starts approximately 2 to 3 cm medial to the AC joint and
extends laterally to the acromial attachment of the AC capsule. The
lateral clavicle is exposed subperiosteally by carefully elevating the
deltotrapezius fascia, periosteum, and AC capsule in one layer starting
at the medial-lateral incision and extending anteriorly and
posteriorly. In most type III injuries, the AC capsule has been avulsed
from the clavicle, leaving the acromial attachment intact. As the
anterior and posterior subperiosteal flaps are being elevated from the
lateral clavicle, the intact acromial attachment of the capsule is
preserved. The specifics to further surgical procedures will be
discussed later in the chapter.
CURRENT TREATMENT OPTIONS
Type I Injury
The type I injury is characterized by sprained but
intact AC ligaments and normal coracoclavicular ligaments. There is no
role for operative management in the acute setting.
Type II Injury
In a type II injury, the AC ligaments are torn and the
coracoclavicular ligaments are stretched but intact. Nonoperatively
treated type I and II injuries may lead to more chronic disability than
previously recognized.29 A recent series reported by Mouhsine et al.107
found that 52% of patients with types I and II injuries remained
symptomatic at an average 6 year follow-up. Radiographic changes at the
AC joint were common.
Nonoperative Treatment
Acutely, all type II injuries are treated
nonoperatively. The arm should be placed in a sling for 10 to 14 days
or until the symptoms subside. This is followed by an early and gradual
rehabilitation program. With this method of treatment, the subluxation
is ignored. Heavy lifting or unprotected contact sports should be
avoided for 8 to 12 weeks to allow complete ligament healing, because a
second injury before complete ligament healing could convert the
subluxation into a complete dislocation. However, an earlier return to
sports may be facilitated by using a protective pad over the AC joint.
Operative Treatment
Persistent pain after a type II AC injury may be the
result of posttraumatic osteolysis of the clavicle, posttraumatic
arthritis, recurrent anteroposterior subluxation, torn capsular
ligaments trapped within the joint, loose pieces of articular
cartilage, or a detached intraarticular meniscus. Meniscal derangement
is uncommon and is characterized by displacement in and out of the
joint like a torn meniscus in the knee. The type of operative treatment
recommended for symptomatic, chronic type II injuries depends on the
cause of the persistent symptoms. Surgical options include isolated
meniscal débridement, distal clavicle excision, and AC reconstruction.
AC reconstruction is only rarely indicated in instances of persistently
painful anterior-posterior instability.
Type III Injury
Throughout recorded medical history, both nonoperative
and operative methods of treatment of the complete AC dislocation have
enjoyed cyclical popularity. During the 1950s, 1960s, and 1970s,
surgical repair gained widespread popularity. In 1974, Powers and Bach117
polled all chairmen of approved residency training programs in the
United States about their treatment of complete AC injuries and
reported the following findings; the majority of program chairmen
treated type III injury by open reduction. Surgical treatment varied,
but 60% used temporary AC fixation and 35% used coracoclavicular
fixation. Nonoperative treatment was rarely advocated. As a result of
these findings, Powers and Bach117 concluded that surgical repair was the most popular method of treatment for complete AC dislocations.
Powers and Bach117 used the classification of AC joint injuries described by Tossy et al.144
A complete dislocation was described as a rupture of the
coracoclavicular ligaments, the AC ligament, the capsule of the joint,
and the fibers of the deltoid and trapezius musculature. This had been
described as a “freefloating clavicle.” This classification does not
allow distinction between complete dislocations with varying degrees of
soft tissue injury, such as disruption of AC ligaments and
coracoclavicular ligaments with and without injury to the surrounding
musculature. It was not until later that Rockwood121
introduced his classification system, distinguishing among the severity
of these injuries. This presents a problem when comparing the older
literature to the new, because in older reports, a complete
dislocation, or Tossy type III injury, likely includes what we now
consider types III, IV, and V together.
In the early 1990s, Cox27,28 performed a study similar to the Powers and Bach117
study of 1974. Cox mailed surveys to two groups of orthopaedic
surgeons: one group of 62 orthopaedic surgeons participating in the
care of athletes on a regular basis, and the second group of 231
chairmen of orthopaedic residency training programs in North America.
Fifty-one of the 59 orthopaedic surgeons in group 1 who responded
(86.4%) preferred nonoperative management of type III AC dislocations.
Of the surgeons, 30 preferred symptomatic treatment over attempts at
manual reduction. Also, 135 of the 187 orthopaedic chairmen who
responded (72.2%) preferred nonoperative management of type III
injuries, and 97 of these advocated symptomatic treatment rather than
attempts at closed manipulation. Most recently, Nissen et al.108
in 2007 presented a similar study that included a mail-in survey sent
to all members of the American Orthopaedic Society for Sports Medicine
(AOSSM) and approved Accreditation Council for Graduate Medical
Education orthopaedic program residency directors. Of the 664
respondents, 81% (71/87 AOSSM members) to 86% (502/577 directors)
continue to treat uncomplicated type III AC separations conservatively.

P.1225

According to Cox27,28 and Nissen et al.,108
the current preference for treatment of type III AC injuries is
nonoperative, involves symptomatic treatment, and represents a shift
toward nonoperative treatment since the Powers and Bach117 survey of 1974.

Many authors have reported successful nonoperative management for the type III AC dislocation.3,5,10,34,50,81,96,116,118,119,139,149,156 Bjerneld et al.10
reported good to excellent results in the vast majority of patients
treated conservatively in both type II and type III (complete)
injuries. Dias et al.34 followed 44
patients with a complete dislocation for 5 years. At that point,
results were satisfactory in all the patients. In 1996, at an average
of 12.5 years after injury, 30 of the original 44 patients were again
reviewed.119 All had a good outcome.
Galpin and colleagues50 compared conservative treatment to surgical treatment with a Bosworth coracoclavicular screw.14
The results of nonoperative treatment were equal if not superior to
those of operative treatment. The nonoperative group had earlier return
to work and sports. In a review of all AC injuries, Post116 stated that almost all type III AC dislocations could be treated without operative intervention. Taft et al.139
compared a group of patients treated nonoperatively with sling, taping,
or a Kenny-Howard sling to a group treated operatively with AC or
coracoclavicular fixation. Subjective ratings of pain and stiffness and
objective ratings of strength and range of motion were similar in both
groups. There was a much higher complication rate in the operative
group. Press and colleagues118 found
benefits to both operative and nonoperative treatment, but earlier
return to work and sports with nonoperative treatment. There were no
significant differences with respect to shoulder range of motion,
manual muscle strength, and neurovascular findings between the two
groups.
Very few prospective, randomized studies of nonoperative
versus operative treatment for AC dislocations have been performed. One
of the few was by Larsen et al.,81
who compared conservative treatment to operative treatment with AC
fixation using two 2-mm threaded Kirschner wires across the joint. The
rehabilitation period was significantly shorter with nonoperative
treatment, and after 13 months there was no difference in the clinical
results. They recommended operative treatment in thin patients who have
a prominent lateral end of the clavicle, in those who do heavy work,
and in patients whose work requires that the shoulder be held in
abduction and flexion quite often.
The other randomized, prospective trial was by Bannister et al.,4
in which nonoperative treatment was compared to fixation with a
coracoclavicular screw. Conservatively treated patients regained
movement significantly more quickly and fully, returned to work and
sports sooner, and had fewer unsatisfactory results than those having
early operation. In patients with a severe dislocation, defined as
having displacement of greater than 2 cm, however, early surgery
produced better results. Bannister et al.4
later established a classification based on radiographic analysis.
Injuries in which the clavicle was displaced 2 cm or more were
associated with detachment of the origin of the anterior deltoid. These
patients benefited from early surgery.
Several studies have documented no significant strength
difference in patients when compared to the opposite side with complete
AC separations who were treated nonoperatively.87,118,142,149,156 Wojtys and Nelson’s156
study is unique in that both strength and endurance were tested.
According to their study, both strength and endurance were comparable
to the noninjured side. However, there was a trend (not statistically
significant) toward a decrease in the expected strength and endurance
advantage on the dominant shoulder in patients whose dominant shoulder
was injured.
In 1997, McFarland et al.96
surveyed 42 team orthopaedists representing all major league baseball
teams to ascertain their treatment for a hypothetical starting rotation
pitcher who had sustained a grade III AC joint separation 1 week before
the start of the season. Twenty-nine (69%) reported they would treat
the injury nonoperatively, while 13 (31%) stated they would operate
immediately. Twenty (48%) of the physicians reported treating a total
of 32 such injuries in baseball players. Twenty (62.5%) were treated
nonoperatively, and 12 (37.5%) were treated operatively. There was not
a major difference in outcome between the two groups.
A possible indication for operative treatment is in the polytrauma patient. A report by Gallay and colleagues49
showed that a displaced AC joint injury in a polytrauma patient has a
greater effect on shoulder function than isolated AC joint injuries
when evaluated by both disease-specific and general health outcomes.
Standard treatment methods may be inadequate in this population.
In summary, the management of the type III AC
dislocation is still somewhat controversial, although the majority of
current reports support nonoperative treatment. When reading the
literature, one must consider that in most comparison studies between
operative and nonoperative treatment the type of surgery varies. Rarely
is there consistency between study groups and methods. In most patients
with type III injuries, excellent functional results can be obtained
with nonoperative management. Younger, more active patients with more
severe degrees of displacement and laborers who use their upper
extremity above the horizontal plane may benefit from operative
stabilization.
Nonoperative Treatment
Numerous methods of nonoperative treatment of a complete
AC dislocation have been reported including adhesive strapping, slings,
bandages, braces, traction, pressure dressings, and plaster casts. Most
commonly, patients are treated with short-term sling support followed
by early range of motion. Devices used to maintain reduction by closed
means are rarely used. They cause the patient much discomfort and have
never proven to maintain the reduction. There is also no correlation
between residual deformity and outcome.139
Nonoperative treatment more commonly consists of short-term (1 to 2
weeks) sling support, symptomatic treatment with nonsteroidal
anti-inflammatory medication, and early mobilization. Schlegel et al.128
prospectively followed a group of acute type III injuries, with 80%
favorable results. Of the 20 patients evaluated 1 year after injury, 4
felt their results were suboptimal. There was no difference in range of
motion in any of the patients. Strength tests revealed equal strength
between sides in all but the “bench press,” which revealed a 17%
decrease on the injured side.
Operative Treatment
Operative management of acute type III injuries is occasionally indicated.83
During the 1800s and early 1900s, practically every conceivable
operation for AC dislocation was performed. These procedures included
AC repairs, coracoclavicular repairs, combined

P.1226

AC
and coracoclavicular repairs, coracoclavicular fusion, and dynamic
muscle transfers using the tip of the coracoid process and attached
conjoined tendon.* Currently, the most popular methods of stabilization are coracoclavicular or intra-articular (i.e., AC) repairs (Table 39-2). Many of the specific procedures used today are combinations or modifications of previously described procedures (Fig. 39-20).

TABLE 39-2 Pros and Cons of Operative Treatment Options for Acromioclavicular Joint Dislocations

Pros

Cons

Intra-articular AC Fixation

Anatomic reduction

  • Hardware failure or migration

  • Distal clavicle osteolysis

Extra-articular Coracoclavicular Repairs

Superior strength of initial fixation (screw)

  • Screw failure

  • Bone resorption secondary to hardware

  • Does not address soft tissue injury

Ligament Reconstruction

  • Anatomic repair

  • No risk of metallic hardware failure or retention

  • Less initial fixations strength

  • Harvest coracoacromial ligament

Intra-articular Acromioclavicular Fixation. Most modern,
commonly used techniques employ some type of coracoclavicular fixation
or reconstruction. However, many methods using intra-articular fixation
are described in the literature. One should be cautious in using these
techniques, as the placement of hardware across the AC joint can be
problematic.
Most authors use small, smooth or threaded Steinmann pins. Lizaur and colleagues85
advocate the use of two 1.8-mm percutaneous Kirschner wires to
stabilize the joint, and they emphasized repair of the damaged deltoid
and trapezius. These pins can be inserted from the lateral edge of the
acromion through the joint and into the clavicle or from the joint out
through the acromion and then back across the joint into the clavicle.
Fama and Bonaga44 reported the use
of a smooth wire inserted laterally in the acromion, through the AC
joint, out the posterior cortex of the clavicle, and through the skin
posterior to the clavicle. The wire was then bent back on itself to
meet the other end of the wire. The two ends were then fastened
together like a safety pin to prevent migration. It must be emphasized
that despite the fact that the pin is bent, it can break, migrate, and
create serious consequences. (The section on complications, below,
discusses pin migration to the spinal cord, lung, subclavian artery,
pulmonary artery, mediastinum, heart, and other areas.)
FIGURE 39-20 Various operative procedures for injuries to the AC joint. A. Suture between the clavicle and the coracoid process. B. Steinmann pins across the AC joint. C. A lag screw between the clavicle and the coracoid process.
A comparison of AC methods of fixation was reported by Eskola and coworkers.41
They performed a prospective, randomized trial in 100 patients using
three types of AC fixation: (a) smooth pins, (b) threaded pins, and (c)
cortical screws. Thirteen of the 86 patients available for review
developed symptomatic osteolysis of the distal clavicle. Eight of the
13 cases of osteolysis occurred among the 25 patients who underwent
cortical screw fixation. In a 4-year follow-up on 70 of the 100 cases,
the results were graded as good in 67 of the 70 patients.42 These authors preferred the use of the threaded Kirschner wires.
Regardless of the method of fixation, the majority of
authors report the use of other procedures as an adjunct to AC
fixation. Neviaser106 introduced
superior AC ligament reconstruction through transfer of the coracoid
attachment of the coracoacromial ligament to the superior aspect of the
clavicle. He did not

P.1227

recommend repair of the coracoclavicular ligament. Ho and colleagues69
advocated reconstruction of the superior AC ligament using the
coracoacromial ligament and reconstruction of the coracoclavicular
ligament using Marlex tubing passed inferior to the coracoid and over
the clavicle. These procedures were done alone or in combination.
Paavolainen et al.113 described AC fixation in combination with AC and coracoclavicular ligament repair.

More recently, a technique involving the use of a hook plate has been described.60,61,132
This technique has a high complication rate, and a second procedure is
always required to remove the plate. This plate does not offer any
benefit over more commonly used procedures.
Extra-articular Coracoclavicular Repairs. The technique
of placing a screw between the clavicle and the coracoid was described
by Bosworth14 in 1941. The screw was placed percutaneously, using local anesthesia and fluoroscopic guidance (Fig. 39-21).
With the patient in a seated position, a stab wound was made on the
superior aspect of the shoulder, 3.8 cm medial to the distal end of the
clavicle. After a drill hole was made in the clavicle, an assistant
reduced the AC joint by depressing the clavicle and elevating the arm
using a special clavicledepressing instrument. An awl was used to
develop a hole in the superior cortex of the base of the coracoid
process, which was visualized using fluoroscopy. A regular bone screw
was then inserted. The screw was left indefinitely, unless specific
indications for removal developed. Bosworth14 did not recommend either repair of the coracoclavicular ligaments or exploration of the AC joint. Bosworth14 also developed a lag screw with a broad head, which he preferred to the original regular bone screw.
Percutaneous insertion of a cannulated coracoclavicular screw was reported by Tsou145
in 1989, who placed a guide pin fluoroscopically from the clavicle to
the coracoid process. After adequate positioning of the pin within the
coracoid had been confirmed radiographically, a cannulated drill bit
and screw were sequentially passed over the guide pin. Tsou145
reported a 32% technical failure rate in 53 patients with complete AC
dislocation using this technique. Accurate insertion of the screw is
difficult to perform percutaneously. Furthermore, the percutaneous
technique does not allow coracoclavicular ligament repair, deltoid and
trapezius reattachment, or AC joint débridement.
FIGURE 39-21
Postoperative anteroposterior radiograph of the shoulder with a
Bosworth screw in place. Note that the AC joint has been reduced and
the coarse lag threads of the screw are well seated into the coracoid
process.
Many other surgeons have reported the use of a Bosworth screw or a slight modification of the original technique.4,50,138 Rockwood’s group59
presented a technique for the chronic, symptomatic dislocated AC joint
in which the coracoacromial ligament was transferred from its acromial
insertion into the medullary canal of the clavicle, along with
temporary placement of a coracoclavicular screw to stabilize the
clavicle until the ligament healed. The screw is usually removed 8
weeks postoperatively, necessitating a second procedure. A
bioabsorbable screw may afford comparable strength to a steel screw,140 and may obviate the need for removal.
Coracoclavicular fixation by methods other than a screw has been reported. Stam and Dawson136 and Goldberg et al.55 described the use of cerclage Dacron ligaments looped over the clavicle and under the coracoid. Verhaven et al.148
utilized a double Dacron velour ligament for fixation in a prospective
study in 28 consecutive patients with a mean follow-up of 5.1 years;
71% had good or excellent results. There was little correlation between
the end result and the degree of residual dislocation, coracoclavicular
ossification and posttraumatic arthritic changes, or osteolysis of the
distal clavicle. Browne and associates17 used 5-mm Mersilene (Ethicon, Somerville, NJ) tape for coracoclavicular fixation. Morrison and Lemos101
reported 12 of 14 good and excellent results when using a synthetic
loop placed through drill holes in the base of the coracoid and the
anterior third of the clavicle.
Cerclage techniques using various synthetic materials have also been described.23,67,68,74,110
Most of these authors also reported AC joint débridement, AC joint
ligament repair or reconstruction, coracoclavicular ligament repair,
and imbrication of the deltotrapezius fascia—either alone or in
combination—in addition to the coracoclavicular fixation.
Ligament Reconstruction. The majority of reconstructive
procedures accepted today involve ligament reconstruction with or
without augmentation with suture, tape, or hardware. The recent
literature has focused on anatomic coracoclavicular reconstruction to
more closely reproduce the intact ligamentous state, thus enhancing
stability.26,57,94,95 Costic et al.26
found that anatomic coracoclavicular reconstruction more closely
approximates the stiffness of the native ligamentous state than does a
standard Weaver-Dunn repair. Mazzocca et al.95
performed a biomechanic cadaver study comparing an anatomic
coracoclavicular ligament reconstruction using a semitendinosis graft
to

P.1228

a
modified Weaver-Dunn procedure and an arthroscopic technique using
nonabsorbable suture material. Specimens were tested with a
directionally applied force (anterior, posterior, and superior) as well
as cyclic loading and load to failure to simulate physiologic states at
the AC joint. These were compared to the intact ligamentous state.
Results showed the anatomical coracoclavicular ligament reconstruction
had significantly less anterior and posterior translation when compared
to the other forms of reconstruction.95 Superior displacement during cyclic loading was equal among the groups.95
They concluded that anatomic ligamentous reconstruction more closely
approximated the intact state, thus providing more stability than the
other two forms of reconstruction. A similar study by Grutter et al.57
also showed superior biomechanic results of anatomic ligament
reconstruction when compared to a modified Weaver-Dunn technique when
specimens were loaded to failure in the coronal plane. The authors
further concluded that anatomic reconstruction recreated the tensile
strength of the intact state when a flexor carpi radialis tendon was
used.57

Type IV and V Injuries
Because of the severe posterior displacement of the
distal clavicle in a type IV injury, and the gross superior
displacement in the type V injury, most authors recommend surgical
repair.107,135
The posterior displacement of the clavicle into the trapezius in a type
IV injury will lead to discomfort with motion. Therefore, surgical
reduction of the deformity and stabilization of the clavicle are often
necessary. The posterior displacement produces significant stripping of
the deltotrapezial fascia from the distal clavicle. Its repair at the
end of the stabilization procedure is a crucial step in augmenting
distal clavicular stability.
Type V injuries require operative reduction and
stabilization because they result in significant deltotrapezial
stripping and resultant gross instability, which commonly leads to
chronic pain and disability. Again, meticulous repair of the
deltotrapezial fascia augments the repair.111
Type VI Injury
All type VI injuries described in the literature have been treated with surgery.54,97,124
Initial attempts at closed reduction failed. In one instance, after
open reduction by lateral retraction of the scapula, the clavicle was
stabilized by suturing the deltoid and trapezius muscle avulsion and by
repairing the AC joint capsule. In one patient, whose shoulder was
operated on 2.5 months after injury, a Steinmann pin was used to
stabilize the AC joint. After immobilization for 3 to 5 weeks, both of
these patients had almost full range of motion and good power. One
patient with a recurrent subcoracoid dislocation was treated by
excision of the distal 1 cm of the clavicle, and at 5-year follow-up
the patient had no complaints or weakness. Gerber and Rockwood54
reported on using the extra-articular technique with a coracoclavicular
lag screw combined with repair of the ligaments and imbrication of the
deltotrapezius fascia over the top of the clavicle.
Type III Variants
AC dislocation with a fracture of the coracoid process
is not a common injury in adults. The mechanism of injury may be
essentially the same as for a type III AC dislocation, except the
coracoclavicular ligaments do not fail and a fracture occurs through
the base of the coracoid, allowing relative upward displacement of the
distal clavicle. It is important to recognize that, in order for the
coracoid fracture to be responsible for the AC displacement, the
fracture must occur through the base. If the fracture occurs at the tip
of the coracoid process, either concomitant coracoclavicular ligament
injury or a second fracture through the coracoid base has occurred,
allowing displacement of the AC joint (see Fig. 39-10).
The isolated coracoid tip fracture, when it occurs in conjunction with
an AC dislocation, is thought to result from a sudden pull on the
coracoid process by the conjoined tendons.150
Coracoid process fractures may not always be easy to
recognize, and it is important that good radiographs be obtained to
look for this injury before surgical management. Although the axillary
radiographic view can suggest a fracture of the coracoid process, the
Stryker notch view or the CT scan can best indicate the site and degree
of displacement of the fracture (see Fig. 39-12).
Placement of an interfragmentary screw across a fracture
of the coracoid base with intact coracoclavicular ligaments is
difficult. Therefore, the need for surgery is one of the few
indications for placement of pins across the AC joint. If the fracture
involves the tip of the coracoid and there is significant displacement,
then screw fixation may be indicated for the coracoid fracture and the
AC joint may be stabilized by a coracoclavicular loop.150
Conversely, fibrous unions of the tip of the coracoid process are often
asymptomatic. Consequently, a combined AC dislocation and fracture of
the tip of the coracoid process may be treated nonoperatively using
methods similar to those prescribed for isolated type III injuries.
Bernard and associates9
reported 4 cases of combined AC dislocation and fracture of the
coracoid base and reviewed 13 cases from the literature. They concluded
that although surgery could produce good results, equally satisfactory
function and minimal residual deformity could be achieved by
immobilization of the shoulder in a sling for 6 weeks. Kumar76 had good short-term success with conservative treatment as well.
Chronic Acromioclavicular Injuries
Patients with types I and II AC joint injuries may develop late degenerative changes.8,29,102
The meniscus and articular cartilage often sustain an injury that leads
to these degenerative changes. Chronic pain after types I and II
injuries is treated with mild analgesics such as nonsteroidal
anti-inflammatory medication, avoidance of painful activity or
positions, and intra-articular injection with corticosteroid
preparations. Many will resolve with this conservative treatment.
Patients with chronic type I injuries who do not respond
to conservative care may require operative excision of the distal
clavicle to provide relief of pain. This can be performed using an open
or an arthroscopic technique.47,51,52,58,103,134
The important aspect of either technique is preservation or repair of
the AC joint capsule to maintain anteroposterior stability of the joint.15
In patients with chronic pain after type II AC sprains,
the initial conservative regimen is the same as for type I injuries. If
conservative, symptomatic treatment fails, surgery may be indicated.
Isolated distal clavicle excision after a type II injury may fail
because of anteroposterior instability of the distal clavicle and
resultant posterior abutment of the clavicle on the scapular

P.1229

spine.
Therefore, the patient should be examined carefully for increased
anteroposterior translation of the clavicle relative to the acromion
during surgical planning. If indeed, anteroposterior instability
exists, distal clavicle excision may be combined with AC capsular
reconstruction or coracoacromial ligament transfer.152

Chronic pain and instability after complete AC
dislocations (types III, IV, and V) should not be treated with isolated
distal clavicle excision. This merely shortens the clavicle without
stabilizing it and is often associated with persistent postoperative
symptoms. Therefore, distal clavicle excision should be combined with
stabilization in chronic, symptomatic, complete AC injuries. The most
popular reconstructive procedure is transfer of the acromial attachment
of the coracoacromial ligament to the resected surface of the distal
clavicle and concurrent coracoclavicular stabilization.
Coracoclavicular stabilization greatly increases the strength of the
construct.36,59,62,63,77,78,118,131,142,151,153
Pearls and Pitfalls
A thorough understanding of coracoclavicular and AC
anatomy is essential to the safe and effective treatment of AC joint
injuries. Table 39-3 lists some tips that may
help assist the surgeon during reconstruction of the AC joint as well
as help to stay away from some of the common pitfalls associated with
this procedure.
COMPLICATIONS
An inferiorly and medially directed force applied to the
dorsum of the acromion most often results in injury to the AC
articulation, the sternoclavicular articulation, or the shaft of the
clavicle. However, combined injuries have been reported. Wurtz and
associates158 and Lancourt80
have reported combined AC dislocation and clavicle fracture. Several
reports exist of simultaneous dislocations of both the sternoclavicular
and AC ends of the clavicle or bipolar dislocations.2,25,39,53,71,127
Bipolar clavicular dislocation (i.e., combined AC and sternoclavicular
dislocation) is a rare injury that has been sparsely reported in the
literature.2,127
When this injury does occur, it is most often a posterior or type IV AC
dislocation associated with an anterior sternoclavicular dislocation.127
This underscores the importance of a thorough evaluation of any patient
with an AC joint injury with particular attention being paid to the
sternoclavicular joint.
Many complications result from operative treatment of AC

P.1238

dislocations. Besides the obvious wound infection and osteomyelitis
that might develop from the operative procedure, several other
complications occur (e.g., a fracture through a drill hole, loss of
purchase of the internal fixation, metal failure, and migration of the
fixation device to other parts of the body). The incidence of recurrent
deformity is probably higher than routinely expected. This should not
be very surprising. There is nowhere else in the body where the weight
of an entire extremity is supported through so little fixation (Fig. 39-27).
In addition, the potential planes of motion at the injured AC joint are
numerous and result in complex loading of the reconstruction. For these
reasons, we advocate longer and more aggressive immobilization than is
commonly described.

TABLE 39-3 Pearls and Pitfalls
  • While exposing the clavicle, create thick, intact deltotrapezial flaps to assist closure at the end of the case

  • Clear the medial and lateral margins of the coracoid to assist passage of suture and graft under it

  • Use a Satinsky vascular clamp to pass the shuttle suture under the coracoid from medial to lateral

  • Keep an adequate bone bridge between the drill holes in the clavicle to reduce the risk of fracture

  • Clear tissue from drill holes on the undersurface of the clavicle to ease passage of the graft

  • Push up on the elbow to reduce the acromion to the clavicle before securing the repair

Migration of Pins
Pins used to stabilize the AC joint have a colorful,
interesting history of migration into remote, life-threatening
locations such as the lung,93 the spinal cord,109 the neck84 posterior to the carotid sheath, and the pleura or close to it.38 Vessels in the thorax and neck have also been the recipients of pin migration injury (Fig. 39-28).
In most instances, pin migration can be prevented by
bending a hook on the portion of the pin that protrudes from the
acromion process. However, the pins can break, and then part of the pin
is free to migrate. Patients must be prepared and forewarned of the
possible necessity of pin removal and the complications of pins that
are not removed.
Lyons and Rockwood86
reviewed 37 reports of pin migration in operations about the shoulder.
They recommended that pin use should be avoided in operations about the
shoulder. When pins are utilized, they should be bent or have
restraining devices to decrease the risk of migration. The patient
should be informed of the risks. Close follow-up should be performed
and the pins should be removed at the conclusion of therapy or whenever
migration is noted. Pin migration may have devastating consequences.
Eight patients have died.
Failure of Soft Tissue Repairs
In the treatment of a ligamentous injury to the AC
joint, the simple repair of the coracoclavicular and AC ligaments
without the additional support of coracoclavicular sutures, screws, or
internal fixation, the AC fixation will likely fail. This is
particularly true in chronic injury to the AC and coracoclavicular
ligaments, and there is often a major separation between the clavicle
and the coracoid. Transfer of the acromial attachment of the
coracoacromial ligament onto or into the medullary canal of the distal
clavicle alone is not strong enough. It must be supplemented with
additional fixation as recommended in the treatment section.
FIGURE 39-27 Radiograph showing failure of fixation of a coracoclavicular screw.
FIGURE 39-28 A broken Steinmann pin has migrated into the right lung field from its previous location in the right AC joint.
Failure of soft tissue repairs can also result from
suture breakage, suture anchor pullout, or screw breakage. If failure
is noticed early in the postoperative period, reoperation to correct
the problem is usually indicated. If failure occurs weeks to months
after surgery, infection should be suspected and ruled out.
Complications Related to Nonabsorbable Tape or Suture
Coracoclavicular fixation using grafts or synthetic
material has been associated with various complications. Goldberg and
colleagues55 recognized erosion of a Dacron graft through the distal clavicle in some cases (Fig. 39-29). Moneim and Balduini100
noted a coracoid fracture after reconstruction of the coracoclavicular
ligaments through two drill holes in the clavicle. Fractures of the
distal clavicle secondary to the use of loop sutures between the
coracoid and the distal clavicle have been reported.37,90 Other complications include aseptic foreign body reactions23 and infections.105 Neault et al.105
reported three cases in which nonabsorbable tape or suture was directly
related to a postoperative infection. Two infections occurred within a
year, but one was 5 years after repair of a type III injury. Colosimo
et al.23 reported two cases of
aseptic foreign body reactions to Dacron graft material 2 and 4 years
after surgery. Microscopic examination revealed chronic

P.1239

inflammation
with a foreign body giant cell reaction. Both patients improved after
removal of the Dacron material and were back to work in 10 days.

FIGURE 39-29
Complications from using cerclage material as a loop around the top of
the clavicle and under the coracoid process for treatment of complete
AC dislocations. A. Preoperative radiograph demonstrating the type III dislocation of the AC joint. B. The postoperative radiograph demonstrates near anatomic reduction using a 6-mm soft Dacron vascular graft loop. C. Gradual erosion of the Dacron graft completely through the distal clavicle can be seen. D. Note the superior migration of the shaft of the clavicle.
Acromioclavicular Arthritis
Symptomatic arthritis of the AC joint may occur after surgical fixation. Weaver and Dunn152
recommended distal clavicle excision and transfer of the coracoacromial
ligament for both acute and chronic AC separations. In a review
article, Cook and Heiner24 recommend
distal clavicle excision as part of the acute surgical management of
patients with AC separations. They reported that postoperative
degenerative changes in the AC joint may be as high as 24% and that
primary excision of the distal clavicle was associated with little
morbidity. Conversely, the vast majority of series reporting the
results of surgical management of AC separations do not advocate
primary excision of the distal clavicle.66
Therefore, there is no consensus regarding primary distal clavicle
excision and coracoacromial ligament transfer for acute AC injuries.
CONTROVERSIES AND FUTURE DIRECTIONS
The Role of Arthroscopy in the Treatment of Acromioclavicular Joint Injuries
Recent literature has proposed an increased role for the
use of arthroscopy in the treatment of AC joint injuries. Wolf and
Pennington155 described an
all-arthroscopic technique of AC joint reconstruction. The coracoid is
visualized through the subcoracoid recess in the anterior aspect of the
joint. An anterior cruciate ligament guide is used to drill a hole
through the clavicle and coracoid, and SecureStrand cable (Surgical
Dynamics, Norwalk, CT) is used for fixation. They reported a series of
4 patients with no recurrence of deformity. Lafosse et al.80
published an all-arthroscopic technique for coracoclavicular ligament
reconstruction, which releases the coracoacromial ligament from the
undersurface of the acromion and transfers it to the inferior clavicle.
Proponents of arthroscopic reconstruction claim the benefits of this
more minimally invasive technique

P.1240

are less violation of deltotrapezial fascia, faster postoperative recovery, less pain, and fewer complications.

Although many different arthroscopic techniques have
been proposed, few clinical outcome studies have been published.
Chernchujit et al.21 published a
series of 13 patients who had an arthroscopic reconstruction following
AC joint disruption using suture anchors and a titanium plate. These
patients were followed for a mean of 18 months. Twelve patients
returned to work without pain within 3 months after the operation and
the average constant score at last follow-up was 95. Postoperative
radiographs confirmed an anatomic reduction in 10 patients, residual
subluxation in two patients, and redislocation of the joint in one
patient. Given this, as well as evolving surgical techniques, we may
see an expanding role for arthroscopy in the future for the management
of these injuries.
REFERENCES
1. Adams FL. The Genuine Works of Hippocrates. Vols 1 and 2. New York: William Wood, 1886.
2. Arenas AJ, Pampliega T, Iglesias J. Surgical management of bipolar clavicular dislocation. Acta Orthop Belg 1993;59:202-205.
3. Bannister
GC, Wallace WA, Stableforth PG, et al. A classification of acute
acromioclavicular dislocation: a clinical, radiological, and anatomical
study. Injury 1992;23:194-196.
4. Bannister
GC, Wallace WA, Stableforth PG, et al. The management of acute
acromioclavicular dislocation. A randomized prospective controlled
trial. J Bone Joint Surg 1989;71B:848-850.
5. Barber FA. Complete posterior acromioclavicular dislocation: a case report. Orthopedics 1987;10:493-496.
6. Bearden JM, Hughston JC, Whatley GS. Acromioclavicular dislocation: method of treatment. J Sports Med 1973;1:5-17.
7. Bearn
JG. Direct observations on the function of the capsule of the
sternoclavicular joint in clavicular support. J Anat 1967;101:159-170.
8. Bergfeld
JA, Andrish JT, Clancy WG. Evaluation of the acromioclavicular joint
following first and second-degree sprains. Am J Sports Med
1978;6:153-159.
9. Bernard
TNJ, Brunet ME, Haddad RJ. Fractured coracoid process in
acromioclavicular dislocations: report of four cases and review of the
literature. Clin Orthop 1983;175: 227-232.
10. Bjerneld
H, Hovelius L, Thorling J. Acromioclavicular separations treated
conservatively. A 5-year follow-up study. Acta Orthop Scand
1983;54:743-745.
11. Black
GB, McPherson JAM, Reed MH. Traumatic pseudodislocation of the
acromioclavicular joint in children: a 15-year review. Am J Sports Med
1991;19:644-646.
12. Blazar
PE, Iannotti JP, Williams GR. Anteroposterior instability of the distal
clavicle after distal clavicle resection. Clin Orthop 1998;348:114-120.
13. Boehm
TC. The relation of the coracoclavicular ligament insertion to the
acromioclavicular joint. A cadaver study of relevance to lateral
clavicle resection. Acta Orthop Scand 2003;74(6):718-721.
14. Bosworth BM. Acromioclavicular separation: new method of repair. Surg Gynecol Obstet 1941;73:866-871.
15. Branch
TP, Burdette HL, Shahriari AS, et al. The role of the acromioclavicular
ligaments and the effect of distal clavicle resection. Am J Sports Med
1996;24:293-297.
16. Breslow
MJ, Jazrawi LM, Bernstein AD, et al. Treatment of acromioclavicular
joint separation: suture or suture anchors? J Shoulder Elbow Surg
2002;11(3):225-229.
17. Browne
JE, Stanley RF, Tullos HS. Acromioclavicular joint dislocations:
comparative results following operative treatment with and without
primary distal clavisectomy. Am J Sports Med 1977;5:258-263.
18. Cadenat FM. The treatment of dislocations and fractures of the outer end of the clavicle. Int Clin 1917;1:145-169.
19. Cahill BR. Osteolysis of the distal part of the clavicle. J Bone Joint Surg 1982;64A: 1053-1058.
20. Carr AJ, Broughton NS. Acromioclavicular dislocation associated with fracture of the coracoid process. J Trauma 1989;29:125-126.
21. Chernchujit
B, Tischer T, Imhoff AB. Arthroscopic reconstruction of the
acromioclavicular joint disruption: surgical technique and preliminary
results. Arch Orthop Trauma Surg 2006;126:575-581.
22. Codman
EA. Rupture of the supraspinatus tendon and other lesions in or about
the subacromial bursa. In: Codman EA, ed. The Shoulder. Boston: Thomas
Todd, 1934: 32-65.
23. Colosimo
AJ, Hummer CD 3rd, Heidt RS Jr. Aseptic foreign body reaction to Dacron
graft material used for coracoclavicular ligament reconstruction after
type III acromioclavicular dislocation. Am J Sports Med 1996;24:561-563.
24. Cook DA, Heiner JP. Acromioclavicular joint injuries. Orthop Rev 1990;14:510-516.
25. Cook F, Horowitz M. Bipolar clavicular dislocation: report of a case. J Bone Joint Surg 1987;64A:145-147.
26. Costic
RS, Labriola JE, Rodosky MW. Biomechanical rationale for development of
anatomical reconstructions of coracoclavicular ligaments after complete
acromioclavicular joint dislocations. Am J Sports Med 2004;32:1929-1936.
27. Cox JS. Acromioclavicular joint injuries and their management principles. Ann Chir Gynecol 1991;80:155-159.
28. Cox JS. Current method of treatment of acromioclavicular joint dislocations. Orthopedics 1992;15:1041-1044.
29. Cox JS. The fate of the acromioclavicular joint in athletic injuries. Am J Sports Med 1981;9:50-53.
30. Curtis RJ. Operative management of children’s fractures of the shoulder region. Orthop Clin North Am 1990;21:315-324.
31. Daly P, Sim FH, Simonet WT. Ice hockey injuries: a review. Sports Med 1990;10: 122-131.
32. Debski
RE, Parsons IM, Woo SL, et al. Effect of capsular injury on
acromioclavicular joint mechanics. J Bone Joint Surg Am
2001;83A:1344-1351.
33. DePalma AF. The role of the disks of the sternoclavicular and the acromioclavicular joints. Clin Orthop 1959;13:222-233.
34. Dias
JJ, Steingold RF, Richardson RA, et al. The conservative treatment of
acromioclavicular dislocation. Review after 5 years. J Bone Joint Surg
1987;69B:719-722.
35. Diaz JJ, Gregg PJ. Acromioclavicular joint injuries in sport: recommendations for treatment. Sports Med 1991;11:125-132.
36. Dumontier
C, Sautet A, Man M, et al. Acromioclavicular dislocations: treatment by
coracoacromial ligamentoplasty. J Shoulder Elbow Surg 1995;4:130-134.
37. Dust
WN, Lenczner EM. Stress fracture of the clavicle leading to nonunion
secondary to coracoclavicular reconstruction with Dacron. Am J Sports
Med 1989;17:128-129.
38. Eaton
R, Serletti J. Computerized axial tomography: a method of localizing
Steinmann pin migration. Orthopedics 1981;4:1357-1360.
39. Echo
BS, Donati RB, Powell CE. Bipolar clavicular dislocation treated
surgically: a case report. J Bone Joint Surg 1988;70A:1251-1253.
40. Eidman DK, Siff SJ, Tullos HS. Acromioclavicular lesions in children. Am J Sports Med 1981;9:150-154.
41. Eskola
A, Vainionpaa S, Korkala O, et al. Acute complete acromioclavicular
dislocation. A prospective randomized trial of fixation with smooth or
threaded Kirschner wires or cortical screw. Ann Chir Gynaecol
1987;76:323-326.
42. Eskola
A, Vainionpaa S, Korkala S, et al. Four-year outcome of operative
treatment of acute acromioclavicular dislocation. J Orthop Trauma
1991;5:9-13.
43. Falstie-Jensen S, Mikkelsen P. Pseudodislocation of the acromioclavicular joint. J Bone Joint Surg 1982;64B:368-369.
44. Fama G, Bonaga S. [Safety pin synthesis in the cure of acromioclavicular luxation]. Chir Organi Mov 1988;73:227-235.
45. Flatow
EL. The biomechanics of the acromioclavicular, sternoclavicular, and
scapulothoracic joints. Instr Course Lect 1993;42:237-245.
46. Flatow
EL, Cordasco FA, Bigliani LU. Arthroscopic resection of the outer end
of the clavicle from a superior approach: a critical, quantitative,
radiographic assessment of bone removal. Arthroscopy 1992;8:55-64.
47. Flatow
EL, Duralde XA, Nicholson GP, et al. Arthroscopic resection of the
distal clavicle with a superior approach. J Shoulder Elbow Surg
1995;4:41-50.
48. Fukuda
K, Craig EV, An KN, et al. Biomechanical study of the ligamentous
system of the acromioclavicular joint. J Bone Joint Surg
1986;68A:434-440.
49. Gallay
SH, Hupel TM, Beaton DE, et al. Functional outcome of acromioclavicular
joint injury in polytrauma patients. J Orthop Trauma 1998;12:159-163.
50. Galpin
RD, Hawkins RJ, Grainger RW. A comparative analysis of operative versus
nonoperative treatment of grade III acromioclavicular separations. Clin
Orthop 1985; 193:150-155.
51. Gartsman GM. Arthroscopic resection of the acromioclavicular joint. Am J Sports Med 1993;21:71-77.
52. Gartsman
GM, Combs AH, Davis PF, et al. Arthroscopic acromioclavicular joint
resection. An anatomical study. Am J Sports Med 1991;19:2-5.
53. Gearen PF, Petty W. Panclavicular dislocation: report of a case. J Bone Joint Surg 1982; 64A:454-455.
54. Gerber
C, Rockwood CA. Subcoracoid dislocation of the lateral end of the
clavicle: a report of three cases. J Bone Joint Surg 1987;69A:924-927.
55. Goldberg
JA, Viglione W, Cumming WJ, et al. Review of coracoclavicular ligament
reconstruction using Dacron graft material. Aust NZ J Surg
1987;57:441-445.
56. Griffiths CJ, Glucksman E. Post traumatic osteolysis of the clavicle: a case report. Arch Emerg Med 1986;3:129-132.
57. Grutter
PW, Petersen SA. Anatomical acromioclavicular ligament reconstruction:
a biomechanical comparison of reconstructive techniques of the
acromioclavicular joint. Am J Sports Med 2005;33:1723-1728.
58. Gurd
FB. The treatment of complete dislocation of the outer end of the
clavicle: a hitherto undescribed operation. Ann Surg 1941;113:1094-1098.
59. Guy
DK, Wirth MA, Griffin JL, et al. Reconstruction of chronic and complete
dislocations of the acromioclavicular joint. Clin Orthop
1998;347:138-149.
60. Habernek
H, Schmid L, Walch G. [Management of acromioclavicular joint
dislocation with the Wolter hook-plate. One year follow-up of 35
cases]. Unfallchirurgie 1993; 19:33-39.
61. Habernek
H, Weinstabl R, Schmid L, et al. A crook plate for treatment of
acromioclavicular joint separation: indication, technique, and results
after 1 year. J Trauma 1993; 35:893-901.
62. Harris
RI, Vu DH, Sonnabend DH, et al. Anatomic variance of the
coracoclavicular ligaments. J Shoulder Elbow Surg 2001;10(6):585-588.
63. Harris
RI, Wallace AL, Harper GD, et al. Structural properties of the intact
and the reconstructed coracoclavicular ligament complex. Am J Sports
Med 2000;28(1): 103-108.
64. Hastings DE, Horne JG. Anterior dislocation of the acromioclavicular joint. Injury 1979; 10:285-288.
65. Havranek P. Injuries of distal clavicular physis in children. J Pediatr Orthop 1989;9: 213-215.
66. Hawkins
RJ, Warren RF, Noble JS. Suture Repair Technique for Acute and Chronic
Acromioclavicular Dislocations. Rosemont, IL: American Academy of
Orthopaedic Surgeons videotape series, 2005.
67. Hessmann
M, Gotzen L, Gehling H. Acromioclavicular reconstruction augmented with
polydioxanonsulphate bands. Surgical technique and results. Am J Sports
Med 1995; 23:552-556.
68. Hessmann
M, Gotzen L, Gehling H, et al. [Results of reconstruction of
acromioclavicular joint rupture with PDS implants]. Unfallchirurg
1997;100:193-197.

P.1241
69. Ho
W-P, Chen J-Y, Shih C-H. The surgical treatment of complete
acromioclavicular joint dislocation. Orthop Rev 1988;27:1116-1119.
70. Inman VT, Saunders M, Abbott LC. Observations on the function of the shoulder joint. J Bone Joint Surg 1944;26:1-30.
71. Jain AS. Traumatic floating clavicle: a case report. J Bone Joint Surg 1984;66B:560-561.
72. Kay SP, Ellman H, Harris E. Arthroscopic distal clavicle excision. Technique and early results. Clin Orthop 1994;301:181-184.
73. Kennedy JC, Cameron H. Complete dislocation of the acromioclavicular joint. J Bone Joint Surg 1954;36B:202-208.
74. Kiefer
H, Claes L, Burri C, et al. The stabilizing effect of various implants
on the torn acromioclavicular joint. A biomechanical study. Arch Orthop
Trauma Surg 1986;106: 42-46.
75. Klimkiewicz
JJ, Williams GR, Sher JS, et al. The acromioclavicular capsule as a
restraint to posterior translation of the clavicle: a biomechanical
analysis. J Shoulder Elbow Surg 1999;8:119-124.
76. Kumar A. Management of coracoid process fracture with acromioclavicular joint dislocation. Orthopaedics 1990;13:770-772.
77. Kumar
S, Sethi A, Jain AK. Surgical treatment of complete acromioclavicular
dislocation using the coracoacromial ligament and coracoclavicular
fixation: report of a technique in 14 patients. J Orthop Trauma
1995;9:507-510.
78. Kutschera
HP, Kotz RI. Bone-ligament transfer of coracoacromial ligament for
acromioclavicular dislocation. A new fixation method used in six cases.
Acta Orthop Scand 1997;68:246-248.
79. Lafosse
L, Baier GP, Leuzinger J. Arthroscopic treatment of acute and chronic
acromioclavicular joint dislocation. Arthroscopy 2005;21:1017-1028.
80. Lancourt
JB. Acromioclavicular dislocation with adjacent clavicular fracture in
a horseback rider: a case report. Am J Sports Med 1990;18:321-322.
81. Larsen
E, Bjerg-Nielsen A, Christensen P. Conservative or surgical treatment
of acromioclavicular dislocation. A prospective, controlled, randomized
study. J Bone Joint Surg 1986;68A:552-555.
82. Lee
KW, Debski RE, Chen CH, et al. Functional evaluation of the ligaments
at the acromioclavicular joint during anteroposterior and
superoinferior translation. Am J Sports Med 1997;25:858-862.
83. Lemos MJ. The evaluation and treatment of the injured acromioclavicular joint in athletes. Am J Sports Med 1998;26:137-144.
84. Lindsey
RW, Gutowski WT. Migration of a broken pin following fixation of the
acromioclavicular joint. Orthopedics 1986;9:413-416.
85. Lizaur
A, Marco L, Cebrian R. Acute dislocation of the acromioclavicular
joint. Traumatic anatomy and the importance of deltoid and trapezius. J
Bone Joint Surg 1994; 76B:602-606.
86. Lyons FA, Rockwood CA. Migration of pins used in operations on the shoulder. J Bone Joint Surg 1990;72A:1262-1267.
87. MacDonald
PB, Alexander MJ, Frejuk J, et al. Comprehensive functional analysis of
shoulders following complete acromioclavicular separation. Am J Sports
Med 1988; 16:475-480.
88. Madsen B. Osteolysis of the acromial end of the clavicle following trauma. Br J Radiol 1963;36:822.
89. Malcapi
C, Grassi G, Oretti D. Posterior dislocation of the acromioclavicular
joint: a rare or an easily overlooked lesion? Ital J Orthop Traumatol
1978;4:79-83.
90. Martell JR. Clavicular nonunion complications with the use of Merselene tape. Am J Sports Med 1992;20:360-362.
91. Martin-Herrero
T, Rodriguez-Merchan C, Munuera-Martinez L. Fracture of the coracoid
process: presentation of seven cases and review of the literature. J
Trauma 1993;30: 1597-1599.
92. Matthews
LS, Simonson BG, Wolock BS. Osteolysis of the distal clavicle in a
female body builder: a case report. Am J Sports Med 1992;21:50-52.
93. Mazet
RJ. Migration of a Kirschner wire from the shoulder region into the
lung: a report of two cases. J Bone Joint Surg 1943;25A:477-483.
94. Mazzocca AD, Arciero RA, Bicos J. Evaluation and treatment of acromioclavicular joint injuries. Am J Sports Med 2007;35:316-329.
95. Mazzocca
AD, Santangelo SA, Johnson ST. A biomechanical evaluation of an
anatomical coracoclavicular ligament reconstruction. Am J Sports Med
2006;34:236-246.
96. McFarland
EG, Blivin SJ, Doehring CB, et al. Treatment of grade III
acromioclavicular separations in professional throwing athletes:
results of a survey. Am J Orthop 1997; 26:771-774.
97. McPhee IB. Inferior dislocation of the outer end of the clavicle. J Trauma 1980;20: 709-710.
98. Meislin
RJ, Zuckerman JD, Nainzadeh N. Type III acromioclavicular joint
separation associated with late brachial plexus neurapraxia. J Orthop
Trauma 1992;6:370-372.
99. Merchan
EC. Osteolysis of the distal clavicle in a woman: case report and a
review of the literature. Ital J Orthop 1992;18:561-563.
100. Moneim
MS, Balduini FC. Coracoid fracture as a complication of surgical
treatment by coracoclavicular tape fixation. A case report. Clin Orthop
1982;168:133-135.
101. Morrison
DS, Lemos MJ. Acromioclavicular separation. Reconstruction using
synthetic loop augmentation. Am J Sports Med 1995;23:105-110.
102. Moushine
E, Garofalo R, Crevoisier X, et al. Grade I and II acromioclavicular
dislocations: results of conservative treatment. J Shoulder Elbow Surg
2003;12(6):599-602.
103. Mumford EB. Acromioclavicular dislocation. J Bone Joint Surg 1941;23:799-802.
104. Murphy OB, Bellamy R, Wheeler W, et al. Posttraumatic osteolysis of the distal clavicle. Clin Orthop 1975;109:108-114.
105. Neault
MA, Nuber GW, Marymont JV. Infections after surgical repair of
acromioclavicular separations with nonabsorbable tape or suture. J
Shoulder Elbow Surg 1996;5: 477-478.
106. Neviaser
JS. Acromioclavicular dislocation treated by transference of the
coracoacromial ligament: a long-term follow-up in a series of 112
cases. Clin Orthop 1968;58:57-68.
107. Nieminen S, Aho AJ. Anterior dislocation of the acromioclavicular joint. Ann Chir Gynaecol 1984;73:21-24.
108. Nissen
CW, Chatterjee A. Type III acromioclavicular separation: results of a
recent survey on its management. Am J Orthop 2007;36;89-93.
109. Norell
H, Llewellyn RC. Migration of a threaded Steinmann pin from an
acromioclavicular joint into the spinal canal. J Bone Joint Surg
1965;47A:1024-1026.
110. Nuber
GW, Bowen MK. Disorders of the acromioclavicular joint:
pathophysiology, diagnosis, and management. In: Iannotti JP, Williams
GR, eds. Disorders of the Shoulder: Diagnosis and Management.
Philadelphia: Lippincott Williams & Wilkins, 1999: 739-764.
111. Nuber
GW, Bowen MK. Disorders of the acromioclavicular joint:
pathophysiology, diagnosis, and management. In: Iannotti JP, Williams
GR, eds. Disorders of the Shoulder: Diagnosis and Management.
Philadelphia: Lippincott Williams & Wilkins, 1999: 739-762.
112. Orava
S, Virtanen K, Holopainen YVO. Posttraumatic osteolysis of the distal
ends of the clavicle: a report of three cases. Ann Chir Gynaecol
1984;73:83-86.
113. Paavolainen
P, Bjorkenheim JM, Paukku P, et al. Surgical treatment of
acromioclavicular dislocation: a review of 39 patients. Injury
1983;14:415-420.
114. Petersson CJ. Degeneration of the acromioclavicular joint. Acta Orthop Scand 1983; 54:434-438.
115. Petersson CJ, Redlund-Johnell I. Radiographic joint space in normal acromioclavicular joints. Acta Orthop Scand 1983;54:431-433.
116. Post M. Current concepts in the diagnosis and management of acromioclavicular dislocations. Clin Orthop 1985;200:234-247.
117. Powers JA, Bach PJ. Acromioclavicular separation: closed or open treatment. Clin Orthop 1974;104:213-233.
118. Press
J, Zuckerman JD, Gallagher M, et al. Treatment of grade III
acromioclavicular separations. Operative versus nonoperative
management. Bull Hosp Jt Dis 1997;56: 77-83.
119. Rawes
ML, Dias JJ. Long-term results of conservative treatment for
acromioclavicular dislocation. J Bone Joint Surg 1996;78B:410-412.
120. Renfree KJ, Riley MK, Wheeler D, et al. Ligamentous anatomy of the distal clavicle. J Shoulder Elbow Surg 2003;12(4):355-359.
121. Rockwood
CA. Injuries to the acromioclavicular Joint. In: Rockwood CA, Green DP,
eds. Fractures in Adults. Vol 1. 2nd ed. Philadelphia: JB Lippincott,
1984:860.
122. Rockwood
CA, Williams GR, Young CD. Injuries to the acromioclavicular joint. In:
Rockwood CA, Green DP, Bucholz RW, Heckman JD, eds. Fractures in
Adults. Vol 2. 4th ed. Philadelphia: Lippincott-Raven, 1996:1341-1414.
123. Rowe CR. In: Cave EF, ed. Fractures and other injuries. Chicago: Year Book Medical; 1961.
124. Sage J. Recurrent inferior dislocation of the clavicle at the acromioclavicular joint. Am J Sports Med 1982;10:145-146.
125. Salter
EG, Nasca RJ, Shelley BS. Anatomical observations on the
acromioclavicular joint and supporting ligaments. Am J Sports Med
1987;15:199-206.
126. Salter EG, Shelley BS, Nasca R. A morphological study of the acromioclavicular joint in humans. Anat Rec 1985;211:353(abst).
127. Sanders JO, Lyons FA, Rockwood CA. Management of dislocations of both ends of the clavicle. J Bone Joint Surg 1990;72A:399-402.
128. Schlegel
TF, Burks RT, Marcus RL, et al. A prospective evaluation of untreated
acute grade III acromioclavicular separations. Am J Sports Med
2001;29(6):699-703.
129. Schmid
A, Schmid F. [Use of arthrosonography in diagnosis of Tossy III lesions
of acromioclavicular joints]. Aktuel Traumatol 1988;18:957-962.
130. Schwarz N, Kuderna H. Inferior acromioclavicular separation: report of an unusual case. Clin Orthop 1988;234:28-30.
131. Shoji
H, Roth C, Chuinard R. Bone block transfer of coracoacromial ligament
in acromioclavicular injury. Clin Orthop 1986;208:272-277.
132. Sim
E, Schwarz N, Hocker K, et al. Repair of complete acromioclavicular
separations using the acromioclavicular-hook plate. Clin Orthop
1995;314:134-142.
133. Skjeldal
S, Lundblad R, Dullerud R. Coracoid process transfer for
acromioclavicular dislocation. Acta Orthop Scand 1988;59:180-182.
134. Snyder S, Banas M, Karzel R. The arthroscopic Mumford procedure: an analysis of results. Arthroscopy 1995;11:157-164.
135. Sondergard-Petersen P, Mikkelsen P. Posterior acromioclavicular dislocation. J Bone Joint Surg 1982;64B:52-53.
136. Stam L, Dawson I. Complete acromioclavicular dislocations: treatment with a Dacron ligament. Injury 1991;22:173-176.
137. Sturm
JT, Perry JFJ. Brachial plexus injuries from blunt trauma—a harbinger
of vascular and thoracic injuries. Ann Emerg Med 1987;16:404-406.
138. Sundaram
N, Patel DV, Porter DS. Stabilization of acute acromioclavicular
dislocation by a modified Bosworth technique: a long-term follow-up
study [published erratum appears in Injury 1992;23(5):359]. Injury
1992;23:189-193.
139. Taft
TN, Wilson FC, Oglesby JW. Dislocation of the acromioclavicular joint.
An endresult study. J Bone Joint Surg 1987;69A:1045-1051.
140. Talbert
TW, Green JR 3rd, Mukherjee DP, et al. Bioabsorbable screw fixation in
coracoclavicular ligament reconstruction. J Long Term Eff Med Implants
2003;13(4): 317-323.
141. Thorndike AJ, Quigley TB. Injuries to the acromioclavicular joint: a plea for conservative treatment. Am J Surg 1942;55:250-261.
142. Tibone
J, Sellers R, Tonino P. Strength testing after third-degree
acromioclavicular dislocations. Am J Sports Med 1992;20:328-331.
143. Tienen
TG, Oyen JF, Eggen PJ. A modified technique of reconstruction for
complete acromioclavicular dislocation. Am J Sports Med 2003;31:655-659.
144. Tossy
JD, Mead NC, Sigmond HM. Acromioclavicular separations: useful and
practical classification for treatment. Clin Orthop 1963;28:111-119.
145. Tsou
PM. Percutaneous cannulated screw coracoclavicular fixation for acute
acromioclavicular dislocations. Clin Orthop 1989;243:112-121.
146. Urist
MR. Complete dislocation of the acromioclavicular joint: the nature of
the traumatic lesion and effective methods of treatment with an
analysis of 41 cases. J Bone Joint Surg 1946;28:813-837.
147. Verhaven
E, Casteleyn PP, De Boeck H, et al. Surgical treatment of acute type V
acromioclavicular injuries. A prospective study. Acta Orthop Belg
1992;58:176-182.
148. Verhaven
E, DeBoeck H, Haentjens P, et al. Surgical treatment of acute type-V
acromioclavicular injuries in athletes. Arch Orthop Trauma Surg
1993;112:189-192.
149. Walsh WM, Peterson DA, Shelton G, et al. Shoulder strength following acromioclavicular injury. Am J Sports Med 1985;13:153-158.

P.1242
150. Wang
K, Hsu K, Shih C. Coracoid process fracture combined with
acromioclavicular dislocation and coracoclavicular ligament rupture: a
case report and review of the literature. Clin Orthop 1994;300:120-122.
151. Warren-Smith
CD, Ward MW. Operation for acromioclavicular dislocation. A review of
29 cases treated by one method. J Bone Joint Surg 1987;69B:715-718.
152. Weaver
JK, Dunn HK. Treatment of acromioclavicular injuries, especially
complete acromioclavicular separation. J Bone Joint Surg
1972;54A:1187-1194.
153. Weinstein
DM, McCann PD, McIlveen SJ, et al. Surgical treatment of complete
acromioclavicular dislocations. Am J Sports Med 1995;23:324-331.
154. Williams
GR, Nguyen VD, Rockwood CR. Classification and radiographic analysis of
acromioclavicular dislocations. Appl Radiol 1989;12:29-34.
155. Wolf EM, Pennington WT. Arthroscopic reconstruction for acromioclavicular joint dislocation. Arthroscopy 2001;17(5):558-563.
156. Wojtys EM, Nelson G. Conservative treatment of grade III acromioclavicular dislocations. Clin Orthop 1991;268:112-119.
157. Wurtz
LD, Lyons FA, Rockwood CA Jr. Fracture of the middle third of the
clavicle and dislocation of the acromioclavicular joint. A report of
four cases. J Bone Joint Surg 1992;74A:133-137.
158. Wurtz
LO, Lyons FA, Rockwood CA. Fracture of the middle third of the clavicle
and dislocation of the acromioclavicular joint. J Bone Joint Surg
1992;74A:133-136.
159. Zanca P. Shoulder pain: Involvement of the acromioclavicular joint: analysis of 1000 cases. AJR 1971;112:493-506.

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